Tumours from argentafinnyh cages. A kartsinoidnyj syndrome

you in section: Criteria of a dysbacteriosis

According to N Mattingly with co-authors (1956), kartsinoidnyj the syndrome developed only in 25% of cases metastazirujushchego kartsinoida that connect with a barrier role of a liver, which intensively metaboliziruet arriving of a tumour serotonin.

Kartsinoidnyj the syndrome is shown by inflow to the top half of trunk. Reddening, sometimes tsianoz separate sites of a skin, in them is thus marked. Geneze inflow, on modern representations, accept uchastieneskolko vazoaktiv th components, including serotonin (E Hegglm, N Laudeman, 1955), bradikinin, kallikrein. In the beginning attacks are rare, and then become frequent; paroxysms are accompanied by haemodynamics infringements (the minute volume of heart decreases, the rhythm is broken, pulse becomes frequent) At patients with kartsinoidom a bronchial tube attacks proceed especially heavy are shown by excitation, anxiety raised potlivostju, slezotecheniem, plentiful salivatsiejnasmorkom. Tgo-visible it is caused to straight lines bystrympostupleniem gumoralnyh agents in a blood channel, passing portal system (A. Kchil with soavt., 1964). At some patients during the period between attacks of inflow the skin keeps tsianotichnyj a shade, in connection with stagnation of blood in the expanded capillaries and small veins, for schoganideskogo porazhyonjja the vascular. Walls (V.V. Menshikov with soavt., 1972J.